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ScarsCoenzyme A and its role in Maintaining Cardiovascular Health 
  Heart disease is the leading cause of death in the United States. Clogged arteries
  (atherosclerosis) can set the stage for life threatening heart attacks.
  What causes clogged arteries? Researchers believe that when LDL cholesterol in the
  bloodstream is oxidized by free radical attack, it tends to build up as plaque on the walls
  of blood vessels, impacting the free flow of blood. To further complicate matters,
  "sticky" blood platelets can clot inside damaged arteries, further cutting off blood flow
  supply to the heart.


  Published research shows that a deficiency of metabolic enzymes exists globally
  specifically the "Master Coenzyme, Coenzyme-A".
  Coenzyme-A is the most active metabolic enzyme in the human body. It operates in the
  body's cells and blood where it initiates over 100 crucial biological functions in the body.
  Coenzyme-A acts as the "universal catalyst"; it is the primary biological cofactor used in
  acyl group transfers. Coenzyme-A initiates the fatty acid metabolism that breaks down
  and degrades the long molecular chains of fatty acids by adding or removing acyl groups.
  This process has significant lipid (fat) lowering activity that inhibits the synthesis of fatty
  acids into cholesterol and triglycerides and it also accelerates the utilization of fatty acids
  as an energy source.

  Further studies show that the problems most people have with metabolizing fat are often
  the  result  of poor nutrition,  which  is  usually  compounded  by  poisoning  from
  environmental toxins.  Coenzyme-A is among the more vulnerable targets for toxic
  metals prescription drugs and fat-soluble environmental toxins. Individuals who have a
  shortage or blockage of Coenzyme-A cannot convert dietary lipids (fats) from animals
  and plants into human fat compositions that can be metabolized and converted to
  essential fatty acids.  If fat cannot be metabolized due to a shortage or blockage of
  Coenzyme-A then the fat simply accumulates. Having accumulated fat deposits that
  cannot be metabolized and converted into energy leads to cardiovascular disease, stress,
  low energy, depression, anxiety, chronic fatigue, obesity and manifestations of other
  disease conditions.

  Coenzyme-A is also required to initiate the tricarboxylic acid (TCA) cycle and
  metabolize fat, carbohydrates, and protein and convert them into cellular energy.  The
  TCA cycle produces more than 90% of the energy the body requires to sustain life.
  Coenzyme-A is constantly being expended by the over demand of metabolic processes of
  the human body and constantly needs replenishing.
  The Most Recent Published Research shows that:

  According to Phyllis A. Balch, CNC and James F. Balch, MD. in the ( THIRD
  EDITION ) of the best selling nutritional guide to natural health "Prescription for
  NUTRITIONAL HEALING." Coenzyme -A, by Coenzyme -A Technologies, Inc.
  Reduces free radical damage to LDL cholesterol and decreases human platelet
  aggregation by supporting the immune systems detoxification of many dangerous
  substances.  It provides an all -natural way to maintain the healthy, normal
  functioning of the heart and the whole cardiovascular system.
  
  In summary, proper nutrition and a healthy diet, combined with Coenzyme-A, a natural
  vitamin and mineral supplement, clean fresh air and exercise, may be the best
  prescription for improving the way your body looks and feels and for providing superior
  "health insurance" for long life.

 REFERENCES:

 1.  Abiko Y.; Metabolism ofCoenzyme-A; New York Academic Press, Third Edition 1975; 7:1-25.
 2.  Knight, G. D., Ph.D.; A Waist is a terrible thing to mind; 1998.
 3.  Leung, L. H., M.D.; Pantothenic Acid as a Weight Reducing Agent: Fasting Without Hunger,
     Weakness and Ketosis; 1995; 44, 403, 405.
 4.  Robishaw, J. D. & Neely, J. R.; Coenzyme A Metabolism; American Journal of Physiology
     1985; 248: El- E9.
5.  Kunz, J. R. M., M.D.; The American Medical Association, Family Medical Guide; Random
     House Inc.; 1982.
6.  Masoro, E. J.; Lipids and Lipid Metabolism; Annual Review of Physiology 1977; 39-301-21.
 7.  Stumpf, P. K.; Metabolism of Fatty Acids; Annual Review of Biochemistry 1969; 38-159-212.
 8.  Krebs, H. A.; The Regulation of Release ofKetone Bodies By the Liver; Advanced Enzyme
     Reaction 1966; 4: 339-354.
9.  Grenville, G. D. & Tubbs, P. K.; The Catabolism of Long-Chain Fatty Acids; Essays in
     Biochemistry 1969; 4-155-212.

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